10、共同机制是-导致肝动脉与门静脉之间的直接交通 ， 引起动脉-门静脉分流 ， 导致肝动脉血流进入门静脉系统 ， 而出现肝动脉血流的重新分配 ， 它是产生肝脏异常高灌注的主要原因,创伤：活检,经皮肝穿刺活检一月后 三角：扇形动脉期一过性强化灶 箭头：肝动脉-门静脉瘘 42-year-old woman with sectorial transient hepatic intensity differences in right hepatic lobe caused by posttraumatic arterioportal shunt. Axial gradient-echo T1-weighted gado 。

11、linium-enhanced arterial phase MR image (TR/TE, 146/2) shows wedge-shaped arterial phenomenon (arrowheads) caused by arterioportal shunt (arrow) due to percutaneous hepatic biopsy performed 1 month earlier,肿瘤： 包括肝脏良性肿瘤与恶性肿瘤 。
肝脏良性肿瘤主要有血管瘤、炎性假瘤及肝局灶性结节增生(focal nodular hyperplasia,FNH)； 肝脏恶性肿瘤主要是指肝细胞癌、胆 。

12、管细胞癌、肝肉瘤及各种富血供转移瘤等 。
恶性病变侵蚀血管,造成动-静脉瘘； 当肿瘤较大伴有门静脉及肝静脉瘤栓形成时,可通过肝窦、脉管等多种途径引起动脉-门静脉、肝动静脉分流 。
肝脏富血供肿瘤的“盗血”作用,也是产生肝脏病理性灌注异常常见原因,65岁男性 ， 肝细胞肝癌； 肝硬化 。
癌灶和子灶 。
门静脉分支癌栓（白色箭头）导致楔形动脉期一过性强化灶 ，癌栓并在T2图像上得到证实,65岁男性患者 ， 肝细胞肝癌； 肝硬化 。
癌灶和子灶 。
门静脉分支癌栓（白色箭头）导致楔形动脉期一过性强化灶 ，并在T2图像上得到证实 。
65-year-old man with liver cirrhosis and hepat 。

13、ocellular carcinoma causing sectorial wedge-shaped transient hepatic intensity difference induced by portal thrombosis secondary to tumor. Axial T2-weighted MR image (12,000/82) confirms portal thrombosis (arrowhead) and shows slight signal intensity changes in triangular area of arterial phenomenon 。

14、 due to small increase in amount of free water,59岁男性患者 ， 结肠癌肝转移 门静脉受压；不是原发的肝动脉血流增加 。
肿瘤外侧的楔形动脉期一过性强化灶； Fig. 10C59-year-old man with large hepatic intraparenchymal metastasis from colon carcinoma and correlated sectorial fan-shaped transient hepatic intensity difference. Axial gradient-echo fat-suppress 。

15、ed T1-weighted unenhanced (146/2) (B) and axial gradient-echo fat-suppressed T1-weighted gadolinium-enhanced arterial phase (146/2) (C) MR images show wide fan-shaped arterial phenomenon with straight border (arrowhead,C) due to hypointense neoplastic lesion at its apex (arrow), causing portal compr 。

16、ession. Note how segment III is also slightly enhanced. Although this transient hepatic intensity difference could look like lobar type because of distribution, this arterial phenomenon is undoubtedly sectorial because lesion, being hypodense and hypoenhancing, causes portal compression and not a pr 。

17、imary increase in arterial flow,50岁男性患者 肝包膜下血管瘤 楔形动脉期一过性强化灶 B超见楔形区域低回声 ，彩色多普勒可见肝动脉-门静脉分流 6B50-year-old man with small round hemangioma beneath Glissons capsule in right hepatic lobe and intralesional arterioportal shunt producing sectorial wedge-shaped arterial phenomenon. Axial T2-weighted (TR/TE,。

18、830/80) (A) and axial gradient-echo unenhanced T1-weighted (216/1.5) (B) MR images show right hepatic lobe nodule (arrow) that is strongly hyperintense inAand hypointense inB,肝包膜下的血管瘤 楔形动脉期一过性强化灶,B超见楔形区域低回声 ，彩色多普勒可见病灶内肝动脉-门静脉分流,34岁女性患者 FNH 虹吸-盗血 1A34-year-old woman with fibronodular hyperplasia in l 。

19、eft hepatic lobe determining homolateral lobar transient hepatic intensity difference (lobar siphoning effect). Axial T2-weighted MR image (TR/TE, 830/80) shows slightly hyperintense nodule (arrow) in left hepatic lobe,34岁女性患者 FNH 虹吸-盗血 病灶周围的强化灶 肝段型实质强化 parenchyma in segments II-IV,34岁女性患者 FNH 虹吸-盗血 。

20、 病灶周围的强化灶 肝段型实质强化 parenchyma in segments II-IV. 门静脉期病灶周围未见强化灶,血管瘤 虹吸-盗血 病灶周围的强化灶 肝段性的强化灶 segment II,胆管炎 多形性 炎性刺激动脉扩张、盗血,T2见扩张的单管 ， 肝周渗出（图1） （图2）管周动脉强化供血 ， 一过性强化 （图3）分布在扩张的胆管旁 ， 假球状强化灶 ， 容易误诊为局部病灶 。
5C57-year-old woman with cholangitis and nonsectorial transient hepatic intensity differences in hepatic dome.。

21、Axial gradient-echo T1-weighted gadolinium-enhanced arterial phase MR images (146/2) show further appearance of arterializations with biliary vessel disease: peribiliary (arrows,B), distributed along dilated biliary vessels, and pseudoglobular, mimicking a focal lesion (arrowhead,C,先天性的肝动脉门静脉瘘,先天性肝动 。

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标题：肝脏|肝脏动脉期一过性强化灶( 二 )